Will scientists be able to replace organs like the colon and/or small intestine eventually for people with IBD?
For example, in the future do you think they will be able to replace a diseased colon that has ulcerative colitis with a new colon grown from the patient’s own extracted stem cells? I do know that they already replace bladders in this way. Also, if you believe this will be possible, when do you think it will happen?
yes, to answerer #2, I am talking about IBD like Crohn’s or Ulcerative Colotis. The question is assuming a cure in the future. One possibility is micobacteria avium paratuberculosis, otherwise known as MAP which has been linked from Johne’s disease in cows. It’s possible that some people with Crohn’s are susceptible to this infection which is prevalent in milk (even after pasteurization) and possibly in cow meat if it hasn’t been cooked well enough. The jury is still out but it’s being looked at closely and researched. Otherwise It could be a possibility for clients who have had their colons removed due to ulcerative colitis, and have been symptom free for awhile.
Eventually, all organs (with the possible exception of the brain) will be replaceable with artificially created ones.
We’ve only been doing this for 32 years, and the science still has a long way to go to replace complex organs like the liver, but I expect to see stem cell generated colons within the next ten years. The ‘small intestine’ is a bit more complicated, so that may still be twenty years or more away.
Histopathology Small intestine–Celiac sprue
how do bacterial and viral infectious diseases transmitted.?
THROUGH THE AIR.
Vestibular Disease bunny rabbit bahnu
Biologists identify a new clue into cellular aging
The ability to combat some age-related diseases may rest with scientists unlocking clues about the molecular and cellular processes governing aging. The underlying theory is that if the healthy portion of an individual’s life span can be extended, it may delay the onset of certain age-related diseases. In the search to understand these molecular processes, researchers at UMass Medical School …
People and Pets: Common Diseases – Epilepsy
Are you one of those people who are affected by the scourge of Gilbert’s disease? Do you feel that you are helpless against the pain and discomfort that it brings? If you do, here are the top three sets of questions that you might want to ask and be answered. Through these, you can get direct answers to questions that hound your mind and can even help you recover-emotionally-as well.
Question number 1: “What causes Gilbert’s disease? Do a viral infection or bacteria cause this condition or can this be passed on in some other way? Is Gilbert’s disease a chronic disease?”
Answer: the abnormal number of the bilirubin in the blood causes Gilbert’s disease, it is not caused by any viral or bacterial infection. It can be passed on through the blood. Gilbert’s disease is known to be hereditary and it remains dormant inside the body for a long time.
Question number 2: “How can you find out if you already have Gilbert’s disease? Are the symptoms of Gilbert’s disease can be associated with jaundice and other liver diseases is this true? How does one find out if his or her condition is Gilbert’s disease? Are there any specific symptoms?”
Answer: You need to be aware of the differences between the symptoms and the signs that are associated with Gilbert’s disease and other liver diseases. Even though, there similarities the differences are also there. Unfortunately, Gilbert’s disease sharing the same symptoms with other diseases is true but if you can look into it further you can spot the minute difference.
Question number 3: “Is there any new method to alleviate, or treat, the symptoms of Gilbert’s disease – yellow eyes, stomach pains and uneasiness? Is it due to the excessive amounts of bilirubin in the blood?”
Answer: You have my understanding. This inherited condition (hereditary) affects just about two to five per cent of the population. It is considered that the situation occurs as a result of troubles in metabolizing the bilirubin. There are more than a few forms of the condition that can be distinguished on biochemical testing. As an outcome, the levels rise and clinical jaundice that you graphically depict occurs.
The condition appears to be not as good as after fasting and during a mild illness. The jaundice then fades instinctively. There is no cure. You ask particularly if there is any new way to reduce the symptoms. I feel sorry that I have been unable to find out if there are any commercial or clinical treatment for Gilbert’s disease.
This condition though is a fairly common sickness mostly amongst the Caucasian population. It’s passed down all the way through families and not frequently considered being life menacing, but ought to still be diagnosed by a medical doctor. The syndrome compromises the body’s capacity to process bilirubin in the liver and causes jaundice. Bilirubin is the yellow substance found in bile. Although the main and most general symptom is jaundice, there are as well quite a lot of other symptoms that you and your physician should be on the lookout for if you think you could have the syndrome.
You have to be very vigilant when it comes to understanding Gilbert’s disease and the knowledge related to it because it changes constantly as doctors learn more about it. be aware that what can be the truth about it today can be different tomorrow.
About the Author
Everything on rules of netball can be found at the Netball Rules website. Visit the Middle Back Pain website to find information on back pain causes. Drop by the Mumps Symptoms website to read about prevention of mumps.
Diseases Gone Wild!
how does Meningococcal meningitis affect the body to cause the symptoms?
hello my question is exactly the what the headline says. i want to no how the bacteria that causes Meningococcal meningitis makes us have the symtoms associated with it. NO THE VIRUS VERSION THE BACTERIA VERSION.
The nasopharynx is the portal of entry through which the bacteria attach with the help of the pili to the epithelial cells of the mucosa . The organism may form part of the nasal flora without producing any symptoms otherwise they may disseminate from the nasal mucosa into the blood stream producing symptoms of like an upper respiratory tract infection . Fulminant meningococcemia later ensues characterised by high fever , hemorrhagic rash , there may be formation of disseminated intravascular coagulation and circulatory collapse ( waterhouse – friderishchen syndrome )
Meningitis is the most common complication of meningococcemia . It begins with headache , vomiting and stiff neck , and progresses to coma in a few hours .
During meningococcemia there is thrombosis of many blood vessels of many organs with perivascular infiltration and petechial hemorrhages . There may be interstitial myocarditis , arthritis , and skin lesion because of this . In meningitis the meninges is acutely inflamed with thrombosis of blood vessels and exudation of polymorphonuclear cells , so the surface of the brain is covered with a thick purulent exufate .
Insidermedicine in 60 – October 10, 2007
